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. 2020 Jun 12;11:894. doi: 10.3389/fphar.2020.00894

Table 3.

Itraconazole and ketoconazole studies included in the systematic review.

Author, year Study population (N) Study type Dose, Duration Sampling times Saliva sampling +
stimulation
Analytical method Saliva
Cmax (mg/L),
AUC0-24
(mg.h/L)
Plasma
Cmax (mg/L), AUC0-24 (mg.h/L)
S/P ratio, correlation S/P ratio based on Support saliva TDM (Y, I, N, N/A)
Itraconazole
Reynes et al. (1997) HIV patients (+/− AIDS),
N = 23
Prospective cohort PK study Suspension, 100mg po bd, 14 days 0, 2, 4, 8 h on day 1 & day 14 syringe Reverse-phase HPLC without AIDS:
1.64 ± 2.05k,
_
with AIDS:
4.07 ± 3.91
_
(no active metabolite detected in saliva)
without AIDS:
0.95 ± 0.38,
AUC0–10:
7.78 ± 3.14
with AIDS:
0.70 ± 0.39,
AUC0–10:
6.25 ± 3.86
Variable C at multiple time-points (day 14) N
Cross et al. (2000) Patients,
N = 40
Randomized, formulation comparison study Suspension,
100mg po bd, 15 days
random, day 15 (median t = 4 h) tubes Reverse-phase HPLC C random times,
0.12 (IQR 0–0.53)
C random times,
0.74 mg/L (IQR 0.46–1.18)
0.115 (0-3.71)l C at multiple time-points, day 15 N
Capsules,
100mg po bd, 15 days
random, day 15 (median t = 3 h) tubes Reverse-phase HPLC not detectedm C random times,
0.61 (IQR 0.37–0.93)
0 C at multiple time-points, day 15 N
Ketoconazole
Force and Nahata (1995) HV,
N = 8
Randomized cross over (with fluconazole) study 400mg po, single
(tablet)
0, 1, 2, 3, 6, 12, and 24 h tubes HPLC 0.119 ± 0.050
_
7.64 ± 3.87
_
0.011 Cmax N

knote high variability (high SD), explains no statistically significant differences between patients with vs. without AIDS; lcalculated from data presented in the study;

mtwo patients had aberrantly high saliva concentrations, which were considered due to sampling or analytical error; C, concentration.