Skip to main content
. 2020 Jun 9;16(6):e1008849. doi: 10.1371/journal.pgen.1008849

Fig 4. AtSCC2 is dispensable for DSB formation but is indispensable for AtSPO11-1-dependent DSB repair.

Fig 4

(A) Localization of γH2AX and DMC1 in WT and Atscc2-5 zygotene male meiocytes. Bar = 5 μm. (B) Plots of the γH2AX and DMC1 foci numbers in WT and Atscc2-5 zygotene male meiocytes (two-tailed Student’s t test). (C) Fluorescence in situ hybridization of WT, Atscc2-5, Atspo11-1-1, Atspo11-1-1 Atscc2-5, Atswi1, and Atswi1 Atscc2-5 double mutant chromosomes using centromere probes. Yellow arrows indicate the separated centromeres of sister-chromatids at metaphase I. Bar = 5 μm.