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. 2020 Jun 19;10:9972. doi: 10.1038/s41598-020-65306-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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An autophagy activator SO ameliorated DSS-induced colitis. C57BL/6J mice were administered either SO or vehicle with (n = 14) or without (n = 7) 2% DSS for 6 days. (a) Body weight changes (means ± SD). (b) Colon length 7 days after starting DSS administration (means ± SD). (c) Representative histopathological images of distal colons (left) and histopathological score (right) (means ± SD from three independent experiments). Data were analyzed by Dunnet’s test, *p < 0.05. Bar; 100 μm. (d) Representative images of TUNEL staining of the colon 6 days after DSS administration. All data are representative of three independent experiments. (e) Concentration of FITC-dextran in the plasma (n = 5 in each group).