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. 2020 Jun 19;11:3130. doi: 10.1038/s41467-020-16915-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Schema showing optogenetic inhibition of the CMT. Certain elements were re-used from a previous publication by the same authors³⁰. b increased δ2-power (right) during NREMS (green) following a 4-h SD in the cingulate (top) and visual (middle) cortices, but not the barrel cortex (bottom) as compared to control SD animals (gray) (two-way rANOVA factors silencing × time; cingulate: F_18,108 = 2.4, p = 0.003; visual: F_18,108 = 4.7, p = 1.8E-07). δ1-power (left) was unaffected in all structures by CMT silencing. c Mean δ1- and δ2-power in these structures in baseline before, during, and after 10 s optogenetic silencing during NREMS of ArchT-expressing CMT neurons (one-way rANOVA factor ‘silencing’; cingulate: F_1,8 = 73.7, p = 3.0E-4; visual: F_1,8 = 48.4, p = 1.0E-4). Red asterisks denote significant post-hoc differences from controls (p < 0.05). Values are expressed as mean ± s.e.m.; in b n = 4, in c n = 6.