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. 2020 May 20;46(2):782–794. doi: 10.3892/ijmm.2020.4611

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Copyright: © Tian et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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AOAA, U0126 and PF-04856264 reduce the increase in excitability of DRGs in SNI rats. (A) Representative traces showing the rheobases of action potentials evoked by current injections to DRG neurons of rats in each group. (B) Typical traces showing the action potentials elicited by twice the strength of the rheobase for 500 msec in DRG neurons of rats in each group. (C) Scatter plot showing the statistical comparison of the rheobase of action potentials in each group. (D) Scatter plot showing the statistical comparison of the number of action potentials elicited by twice the strength of the rheobase for 500 msec in each group. ^*P<0.05. n=8 for each group. DRG, dorsal root ganglion; SNI, spared nerve injury; AOAA, O-(carboxymethyl)hydroxylamine hemihydrochloride. DRG, dorsal root ganglion; SNI, spared nerve injury; AOAA, O-(carboxymethyl) hydroxylamine hemihydrochloride.