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. 2020 Jun 23;10:10149. doi: 10.1038/s41598-020-66797-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Gene ontology analysis of differentially methylated (DM) promoters in KRAS- inhibited pancreatic cancer cells. (A,B) Gene Ontology analysis of DM genes in cells with (A) responsive or (B) refractory DNA methylation. Processes related to development and differentitation are in bold. (C) Venn diagram showing the number of biological processes associated with responsive or refractory promoter CpG methylation in KRAS-depleted cell lines. (D) (Top) List of affected biological processes exclusive to cell lines responsive to KRAS-depletion, or (Bottom) common among all of the KRAS-depleted cell lines.