Table 6.
Percent change of quantitative PCR data from Figs. 4 and 5
| Time (Percent Change), % |
Genotype (Percent Change), % |
|||
|---|---|---|---|---|
| Gene | Male | Female | Male | Female |
| ATP4A | −59* | −51* | −32* | −35* |
| ATP12A | −74* | −71* | 25 | 67 |
| KCNJ1 | 119* | 79* | 41 | 24* |
| KCNJ15 | −43* | −78* | −30* | 13 |
| KCNMA1 | −10 | −72* | −17 | −2 |
| SCNN1A | −12 | −8 | 0.5 | 7 |
| SCNN1B | −41* | −57* | −6 | 37* |
| SCNN1G | −27* | −38* | −11 | 23* |
| SGK1 | −4 | −27 | −26* | −15 |
| SLC12A1 | −11 | 68* | 46 | 3 |
| SLC26A4 | −33 | −90* | −40* | 11 |
| WNK1 | −52* | −43* | 6 | −1 |
| WNK4 | −11 | 19* | 22* | −3 |
Values are shown as percentages. The relative percent change of gene expression for midnight (vs. noon) (time) and knockout (vs. control) (genotype) are given for each sex. ATP4A and ATP12A, gastric and nongastric subunits of H+/K+-ATPase; KCNJ1, gene that encodes renal outer medullary K+ channel protein; KCNJ15, inwardly rectifying K+ channel; KCNMA1, large-conductance channel subunit coding gene; SCNN1A, SCNN1B, and SCNN1G, α-, β-, and γ-subunits of the epithelial Na+ channel, respectively; SGK1, serum/glucocorticoid-regulated kinase 1; SLC12A1, Na+-K+-2Cl− cotransporter gene; SLC26A4, pendrin gene; WNK, with no lysine protein kinase.
Comparisons that were significant using original values.