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. 2020 May 26:aqaa091. doi: 10.1093/ajcp/aqaa091

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© American Society for Clinical Pathology, 2020. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com

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(Case 1) Postmortem microscopic examination of the lungs showed diffuse alveolar damage characterized by hyaline membrane formation (A, ×100) and scattered squamous metaplasia of distal airways (B, ×100) on a background of emphysematous changes. Bronchial sampling showed only minimal submucosal chronic inflammation with unremarkable overlying respiratory epithelium (C, ×100), and the left lower lobe lung mass lesion was consistent with large cell carcinoma (D, ×100). The kidneys were notable for acute tubular injury on a background of moderately extensive chronic changes of the renal parenchyma (E, ×100). The heart showed sequelae of coronary artery atherosclerosis and chronic ischemic injury with replacement-type interstitial fibrosis (F, ×20).

(Case 1) Postmortem microscopic examination of the lungs showed diffuse alveolar damage characterized by hyaline membrane formation (A, ×100) and scattered squamous metaplasia of distal airways (B, ×100) on a background of emphysematous changes. Bronchial sampling showed only minimal submucosal chronic inflammation with unremarkable overlying respiratory epithelium (C, ×100), and the left lower lobe lung mass lesion was consistent with large cell carcinoma (D, ×100). The kidneys were notable for acute tubular injury on a background of moderately extensive chronic changes of the renal parenchyma (E, ×100). The heart showed sequelae of coronary artery atherosclerosis and chronic ischemic injury with replacement-type interstitial fibrosis (F, ×20).