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RSV infected airway epithelial cells (AECs) trigger the production of uric acid, IL-33, TSLP, and CCL2. AECs and macrophages increase the expression of innate cytokines when uric acid is upregulated. CCL2 can recruit monocytes, APC, and T cells to the lung, while IL-33 along with TSLP and IL-1β will recruit and activate ILC2.
