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. 2020 Jun 26;27:11. doi: 10.1186/s40709-020-00122-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Elevated expression of CCND2 AS1 suppressed tumor growth in vivo. a Images of tumors formed in the nude mice injected with the indicated cells. The control or HeLa-pCCND2 AS1 were injected into the right flanks (n = 5), respectively. b, cCCND2 AS1 up-regulated significantly inhibited tumor growth in tumor weights and sizes in nude mice. Tumor volumes were calculated by the formula: V = 0.5 × L × W² after injection every week. Bars indicate SD. Asterisk(s) indicates a significant change (*p < 0.05, **p < 0.01). Data are the mean ± SD