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. 2020 Jun 28;29(9):105095. doi: 10.1016/j.jstrokecerebrovasdis.2020.105095

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Fig 2 — First MRI (A to C). Diffusion-Weighted MRI (DWI) shows a high signal on b 1000 (A) with low Apparent Diffusion Coefficient (ADC) in the occipital lobes (B). Time of Flight (TOF) (C) shows P3 segments of posterior cerebral arteries (PCA) bilateral occlusion (arrows) (C). Second MRI (D to F). TOF shows better visualisation of distal segments of bilateral PCA (arrows) (D). Fluid–attenuated inversion recovery (FLAIR) shows a hypersignal in the initial ischemic lesions (initial FLAIR was normal) (E). Susceptibility-Weighted imaging (SWI) shows hypo-intensity (haemorrhage) concerning the totality of the ischemic lesion (F). MRI perfusion shows an increase cerebral blood volume (CBV) in the right thalamus (arrow) (G) and an increase of MTT in the right hemisphere (H).