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. 2020 Jun 29;10:10588. doi: 10.1038/s41598-020-67448-x

Table 2.

Predictors of parenchymal hematoma: multivariate analysis.

PH at follow-up MRI OR (95% CI); p OR (95% CI); p
Model A Model B
VLCBV regions 2.816 (1.146–6.918), p = 0.024 1.592 (1.133–2.237), p = 0.007
Pretreatment glucose (per IQR) 1.049 (0.755–1.456), p = 0.777 1.072 (0.771–1.491), p = 0.678
Baseline NIHSS (per IQR) 1.337 (0.948–1.887), p = 0.098 1.260 (0.885–1.795), p = 0.200
Rescue MT (vs primary) 1.395 (0.668–2.916), p = 0.376 1.415 (0.674–2.971), p = 0.359
Recanalization status
 > 4′5 h from stroke onset (vs < 4′5 h) 6.656 (2.444–18.130), p < 0.001 7.552 (2.745–20.779), p < 0.001
 No recanalization (vs < 4′5 h) 2.848 (0.867–9.351), p = 0.084 2.810 (0.865–9.132), p = 0.086
Cardioembolic etiology (vs no) 2.584 (1.197–5.577), p = 0.016 2.812 (1.285–6.153), p = 0.010
Sex (females vs males) 0.500 (0.228–1.097), p = 0.084 0.522 (0.237–1.150), p = 0.107
Good collaterals (vs poor) 1.256 (0.550–2.867), p = 0.588 1.369 (0.599–3.129), p = 0.456

Very low cerebral blood volume (VLCBV) was included as a dichotomic variable (yes vs no) in model A, and as a continuous quantitative variable (estimations per IQR of VLCBV increase) in model B. MT: Mechanical thrombectomy; NIHSS: National Institutes of Health Stroke Scale; PH: parenchymal hematoma. The Hosmer–Lemeshow test showed an adequate goodness-of-fit of the final models (Model A: X2 = 4.028, p = 0.855; Model B: X2 = 9.329, p = 0.315), and the models classified correctly a total of 84% (Model A) and 85% (Model B) of cases.