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. 2020 Jun 26;19:1533033820928143. doi: 10.1177/1533033820928143

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — Expression of miR-590 negatively correlates to the expression of HMGA2 in PDAC samples. A, Data from Starbase 2.0 showed that HMGA2 significantly increased in PDAC tissues when compared to normal tissues. B, Data from Starbase 2.0 showed that patients with high expression of HMGA2 had a poor overall survival time compared to patients with low HMGA2 expression. C Database revealed a lower expression of miR-590 in PDAC samples than in paired normal samples. D, The expression of miR-590 was negatively correlated with HMGA2 expression. E, Results from qRT-PCR showed HMGA2 was highly expressed in PDAC tissues than normal group. F, Results from qRT-PCR showed miR-590 was significantly downregulated in PDAC group than normal group. Data are presented as means ± SD of 3 independent experiments (PAAD, Pancreatic adenocarcinoma; *P < .05 compared with control). HMGA2 indicates High mobility group AT-hook 2; miR, microRNA; PDAC, pancreatic ductal adenocarcinoma; qRT-PCR, quantitative real-time polymerase chain reaction; 3′UTR, 3′untranslated region.