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. 2020 Jun 6;18:215–229. doi: 10.1016/j.omtm.2020.05.027

Table 2.

circRNAs Involved in Cardiovascular Diseases

Diseases circRNAs Method +/− Axis Effects Samples Ref.
IMI circNCX1 Sanger sequencing + circNCX1-miR-133a-3p-CDIP1 promotes cardiomyocyte apoptosis fetal cardiomyocyte-derived H9c2 cell 87
MF circRNA_010567 circRNA-array + circRNA_010567-miR-141-TGF-β1 promotes Col I, Col III, and α-SMA expression diabetic mice myocardium and CFs treated with Ang II 88
MF circRNA_000203 circRNA-array + circRNA_000203-miR-26b-5p-Col1a2 and CTGF promotes the expression of Col1a2 and CTGF and promotes myocardial fibrosis myocardium of the db/db mice 31
CH and HF HRCR/(mm9-circ-012559) N/A HRCR-miR-223-ARC overexpression of HRCR can decrease cardiomyocyte size, WH/WB, cardiac stress gene expression, and interstitial fibrosis mouse heart 84
AMI circRNA_081881 circRNA-array circRNA_081881-miR-548-PPARγ as a potential target for the diagnosis and treatment of AMI plasma of AMI patients 89

IMI, ischemic myocardial injury; MF, myocardial fibrosis; CFs, mouse cardiac fibroblasts; CH and HF, cardiac hypertrophy and heart failure; WH/WB, heart weight to body weight ratio; AMI, acute myocardial infarction; circRNA-array, circRNA microarray; N/A, not available; +, upregulate; −, downregulate.