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. 2020 Jun 30;10:10665. doi: 10.1038/s41598-020-67812-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Characterization of mitochondrial acetylation in WT and GCN5L1 cKO animals. (A) Genotype and breeding scheme of WT and GCN5L1 cKO transgenic mice. (B) Diet and tamoxifen (Tam) injection schedule of WT and GCN5L1 cKO mice. (C, D) Overall acetylation of mitochondria isolated from cardiac tissues was greatly increased in HFD WT animals, which was significantly attenuated in HFD cKO mice. Values are expressed as means ± SD, n = 5, *P < 0.05 versus WT LFD, ^$P < 0.05 versus cKO LFD, ^#P < 0.05 versus WT HFD.