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. 2020 Jun 30;10:10618. doi: 10.1038/s41598-020-67457-w

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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OMA1 causes proteolytic cleavage of OPA1 induced by p32/C1QBP deficiency. (a) Wild type, OMA1^−/−, YME1L^−/−, OMA1^−/−YME1L^−/−, and p32^−/− MEFs were analyzed by immunoblotting for the indicated proteins. (b) Wild type and p32^−/− MEFs were transfected with control or two independent siRNA for OMA1 (OMA1-1 and OMA1-2) and subjected to western blotting for the indicated proteins. (c) Wild type, OMA1^−/−, and p32^−/− MEFs were transfected with indicated siRNA. Cell lysates were subjected to Western blotting for indicated proteins.