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. 2020 Jul 1;34(13-14):898–912. doi: 10.1101/gad.336024.119

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© 2020 Pérez-Calero et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 6. — Genome-wide chromatin-associated action of UAP56. (A) Representative screenshot of a genomic region in which UAP56 ChIP-seq (green), PRO-seq (light blue), RNAPII ChIP-seq (red), RNAPII-S5P ChIP-seq (orange), and RNA-seq (magenta) in K562 cells is shown. (B) Venn diagram showing the overlap between protein-coding genes transcribed in K562 cells detected by PRO-seq (light purple) and UAP56 chromatin-bound protein-coding genes detected by ChIP-seq (green). P < 0.001, hypergeometric test. (C) Metaplot analysis of the distribution of the UAP56 ChIP-seq signal (IP-Input) along a model gene body.