| • Postprandial hyperglycaemia is defined as having postprandial glucose level higher than the target after usual meal and on medications (if any) |
| • PPG should be measured 2-h after a usual meal and medications (if any). |
| • Target PPG: 160 mg/dL as long as hypoglycaemia is avoided. |
| • Both non-pharmacologic and pharmacologic therapies should be considered |
| ▫ MNT: diet with low glycaemic load is recommended |
| ▫ AGIs (acarbose, miglitol or voglibose), DPP4 inhibitors, SGLT2 inhibitors or GLP-1 analogues (preferably short acting) as the first add on to metformin therapy |
| ▫ Glinides and short acting sulfonylureas as alternative options |
| ▫ Rapid acting insulin analogues may be considered over the regular insulin when postprandial hyperglycaemia is a concern especially when the risk of hypoglycaemia is high. |
| • Combination therapy of AGI with other agents may be considered. |
| • SMBG should be considered as it is the most practical method for monitoring postprandial glycaemia. |
| • Efficacy of treatment regimens should be monitored frequently to guide therapy towards achieving PPG targets. |