Skip to main content
. 2020 Jun 1;10(16):7193–7210. doi: 10.7150/thno.44877

Figure 7.

Figure 7

Synergistic regulation of miR-1291-ERRα-CPT1C signaling on tumor. (A) WST-8 and BrdU assays were performed to examine the effect of low CPT1C expression on the viability and proliferation capacity of WT and ST-miR1291 tumor cells. Data are mean ± SD (n = 5). (B) Glycolysis inhibition tests with 2-deoxyglucose and glucose deprivation tests with glucose were performed to measure the depletion of CPT1C expression on the anti-metabolic stress ability of WT and ST-miR1291 tumor cells. Data are mean ± SD (n = 5). (C) WST-8 and BrdU assays were performed to examine the influence of ERRα inhibition on the viability and proliferation capacity of WT and ST-miR1291 tumor cells. Data are mean ± SD (n = 5). (D) Glycolysis inhibition tests with 2-deoxyglucose and glucose deprivation tests with glucose were performed to measure the impact of reduction of ERRα expression on the anti-metabolic stress ability of WT and ST-miR1291 tumor cells. Data are mean ± SD (n = 5).