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. 2020 Jun 25;10:1080. doi: 10.3389/fonc.2020.01080

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Copyright © 2020 Kong, Yao, Ding, Dong, Wu, Sun and Zheng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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IF1 attenuated sensitivity of HCC cells after insufficient RFA to sorafenib in vitro. (A,B) The effect of sorafenib on wound-healing capacity of Hep3B, Hep3B-IF1, and Hep3B-H or Hep3B, Hep3B-H, and Hep3B-H-shRNA-IF1 was shown. (C,D) The effect of sorafenib on migration of Hep3B, Hep3B-IF1, and Hep3B-H or Hep3B, Hep3B-H, and Hep3B-H-shRNA-IF1 was shown. (E,F) The influence of sorafenib on migration and tube formation of TAECs-shRNA-IF1 and TAECs was exhibited. (G) The statistical results of (F,G) were shown. (H) The statistical results of (A–D) were shown. (I) The immunohistochemical staining of IF1 in HCC tissue with sorafenib therapy. (J) Kaplan-Meier analysis of DFS and OS for the expression IF1. Data are the means ± SD. *P < 0.05, **P < 0.01, ***P < 0.001; ns, no significance.