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. 2020 Jul 2;20:185. doi: 10.1186/s12890-020-01218-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Overexpression of POU3F3 promoted cancer cell proliferation through miR-30d-5p. Overexpression of POU3F3 mediated enhanced, while overexpression of miR-30d-5p mediated inhibited proliferation of both NCI-H23 and NCI-H522 cell lines. In addition, overexpression of miR-30d-5p attenuated the effects of overexpressing POU3F3 on cancer cell proliferation (*, p < 0.05)