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. 2020 Jul 2;10(3):2045894020936660. doi: 10.1177/2045894020936660

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© The Author(s) 2020

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 1. — Plausible mechanisms of development of right ventricular dysfunction in COVID-19. Schematic showing development of right ventricular dysfunction from COVID-19-associated acute respiratory distress syndrome (ARDS), pulmonary embolism, cytokine storm, micro- and macro-vascular dysfunction, and direct angiotensin-converting enzyme 2 (ACE2)-mediated effects of SARS-CoV-2 virus on the right ventricle.

Note: Figure created with BioRender.com