Figure 10. Schematic diagram illustrates the potential for EV-enriched miRNA27a* to contribute to cardiac hypotrophy through intercellular communication with cardiac fibroblasts.
Under cardiac stress, cardiac fibroblasts secrete EV-enriched miRNA-27a* and/or miRNA-21*, which are transported to cardiomyocytes and regulates the expression of PDLIM5, leading to cardiac hypertrophy.