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. 2020 Mar 28;36(7):685–695. doi: 10.1007/s12264-020-00486-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Impairment of pinprick-evoked information transmission in conditional Nfia null mice. A, B, D, E Representative images of pinprick-induced c-Fos expression in the ipsilateral and contralateral dorsal horn of adult control (A, D) and Nfia^F/F;Wnt1-Cre mice (B, E) (scale bar, 100 µm). C Numbers of c-Fos⁺ neurons in the ipsilateral and contralateral dorsal horns from mice with indicated genotypes and conditions (n = 6/group, one-way ANOVA with Tukey’s post-hoc analysis, F_{2, 15} = 420.2, ****P < 0.0001). F Numbers of c-Fos⁺ neurons in the contralateral dorsal horns from mice with indicated genotypes and conditions (n = 6/group, one-way ANOVA with Tukey’s post-hoc analysis, F_{2, 15} =1.78, P = 0.2024).