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. 2020 Jun 12;21(12):4200. doi: 10.3390/ijms21124200

Figure 2.

Figure 2

Events depicting the role of mitochondrial defects, reactive oxygen species (ROS)-released ROS and mitochondrial and sarcolemmal KATP channels in the generation of ventricular arrhythmias. ROS—reactive oxygen species; sarcKATP—sarcolemmal KATP channels and mitoKATP—mitochondrial KATP channels.