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. 2020 Jul 11;20:303. doi: 10.1186/s12935-020-01395-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — MRTF-A plays a positive role in the regulation of miR-155 expression in GC cells. miR-155 expression was decreased by the selective MRTF-A inhibitor CCG-1423. a, b CCG-1423 inhibited miR-155 expression in SGC-7901 and MGC-803 cells. a–c Cells were treated with 10 μM CCG-1423 for 24 h. RNAs were examined by RT-qPCR. c CCG-1423 inhibited miR-155 promoter activity in SGC-7901 cells. miR-155 expression was stimulated by Cytochalasin D. d, e Cytochalasin D stimulated miR-155 expression in SGC-7901 and MGC-803 cells. d–e Cells were treated with 10 μM Cytochalasin D for 24 h. RNAs were examined by RT-qPCR. c Cytochalasin D increased miR-155 promoter activity in SGC-7901 cells. Data are shown as the mean ± S.E.M, based on three to six independent experiments. **, p < 0.01