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. Author manuscript; available in PMC: 2020 Jul 12.
Published in final edited form as: J Immunol. 2020 Jan 27;204(5):1242–1254. doi: 10.4049/jimmunol.1901191

Table 2.

HIV latency reversal induced by immune checkpoint blockade or latency reversing agents in vitro.

Immune checkpoint blockers
Non-proliferating Proliferating
No SEB + SEB No SEB + SEB
aPD-1 23.4 63.3 31.5 22.4
Isot ctrl aPD-1 25.3 33.8 39.6 43.2
aCTLA-4 38.6 66.3 39.3 53.2
Isot ctrl aCTLA-4 27.0 52.3 47.8 37.1
aPD-1+aCTLA-4 33.2 75.2 42.9 34.2
Isot ctrl 2 ICB 21.6 39.4 41.9 37.2
aPD-1+aCTLA-4 +aTIM-3 40.5 65.3 49.6 40.7
Isot ctrl 3 ICB 22.9 30.2 31.7 36.8
aPD-1+aCTLA-4 +aTIM-3+aTIGIT 44.6 74.1 48.2 35.1
Isot ctrl 4 ICB 22.8 56.8 19.2 39.8
Other LRAs
Non-proliferating Proliferating
DMSO 7.0 2.7
Romi 1 nM 1.2 6.3
Romi 0.1 nM 5.1 7.2
JQ1 1 μM 2.7 2.0
JQ1 0.1 μM 5.2 7.8
Bryo 10 nM 50.9 42.6
Bryo 1 nM 64.8 47.4
PMA+iono 26.5 87.2

The percentage of maximal response induced following stimulation with anti-CD3 and anti-CD28 is shown. Increase in EGFP expression (latency reversal) is shown as the mean % of maximal activation with anti-CD3/28+IL-7+IL-2 following addition of IC blocking (ICB) antibodies or isotype control (isot ctrl), alone or in combination (top panels) or latency reversing agents (LRA; bottom panels). A grey box indicates a significant increase compared to isotype control (for immune checkpoint blockade or DMSO (for other LRAs), defined as p<0.05, using the frequency of EGFP+ cells shown in Figure S3. a = anti; Romi = romidepsin, bryo = bryostatin; PMA = phorbol 12-myristate 13-acetate; iono = ionomycin.