Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 May 26;8(6):802. doi: 10.3390/microorganisms8060802

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

Extra-intestinal pro-inflammatory mediator secretion following NAP treatment of C. jejuni infected secondary abiotic IL10^−/− mice. On day (d) 0 and d1 secondary abiotic IL10^−/− mice were perorally infected with C. jejuni strain 81-176 by gavage and treated intraperitoneally with either synthetic NAP (open circles) or placebo (PLC; closed circles) from d2 until d5. On d6 post-infection, nitric oxide (A,D,G), IFN-γ (B,E,H), and TNF (C,F,I) concentrations were measured in ex vivo biopsies taken from liver (A–C), kidney (D-F), and lung (G–I). Naive mice served as negative controls (open diamonds). Medians (black bars), levels of significance (p-values) as determined by the Kruskal–Wallis test and Dunn’s post-correction, as well as numbers of analyzed mice (in parentheses) are indicated. Data were pooled from four independent experiments.