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. 2020 Jul 7;11:742. doi: 10.3389/fphys.2020.00742

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Copyright © 2020 Su, Fan, Ling, Fan, Xia, Liu, Li, Zhang, Zeng and Tang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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APs activate the AMPKα signal pathway to suppress VSMC proliferation. (A,B) The phosphorylation of AMPKα, Src and Jun were assessed by proteomics analysis of phosphokinase arrays in control (Ctrl) VSMCs and APs cocultured with VSMCs. The phosphorylation of (C) AMPKα, (D) Src, and (E) Jnk was determined with western blot analysis in control (Ctrl) VSMCs, RPs, and APs cocultured with VSMCs. Data are presented as mean ± SD (n = 6). Comparisons between more than two groups were analyzed using one-way ANOVA analysis with the Dunnett’s multiple correction. **P < 0.01 vs Ctrl (VSMCs without platelets).