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. 2020 Jul 15;39:135. doi: 10.1186/s13046-020-01634-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Expression level of THOC1 is closely related to the proliferation of HCC. a Representative immunohistochemistry images of THOC1 in normal cells and tumors cited from the Human Protein Atlas. b Statistical analysis of THOC1 expression level in normal and tumor cells based on the LIHC samples in The Cancer Genome Atlas (TCGA) database (Student’s t test; ***P < 0.001). Statistical analyses of THOC1 expression level in LIHC samples based on (c) pathology grade (one-way ANOVA; **P < 0.01) and (d) clinical stage (one-way ANOVA; **P < 0.01). e Overall survival and (f) disease free survival analysis separated by low and high THOC1 expressions in LIHC samples. g GSEA based on THOC1 expression. (h) Correlation analysis of THOC1 with PCNA and Ki67. **P < 0.01, ***P < 0.001