TABLE 2.
Conceptually, the AAR is the myocardium downstream to an occluded coronary artery that becomes ischemic (1). |
Infarct size and AAR should be ideally measured at the same time point. If measured at different time points, salvage quantification might show implausible results (e.g., negative values) (14–16,90). |
LGE grey zones should not be interpreted as dead, since they likely represent a mixture of bright (dead) and dark (alive) myocardium. Grey myocardium can be due to partial volume effect (15,31). |
Edema development should be interpreted as a manifestation of myocardial I/R injury and should not be considered as a reliable marker of AAR* (15,44,89). |
T1W is the recommended methodology for LGE imaging after MI (2,4). |
T2-mapping is the recommended methodology for edema imaging after MI (3,35). |
T1W LGE is the recommended methodology for MVO imaging (hypointense areas within the LGE area) (95). |
T2* mapping is the recommended methodology for IMH imaging after MI (3). |
Controversial topic (majority but not all panelists in agreement).
AAR = area at risk; LGE = late gadolinium enhancement; other abbreviations as in Table 1.