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Illustration of the oncogenic role of lncRNA AGPG in driving p53‐deficient ESCC development. Loss of p53 promotes AGPG expression, which in turn forms a complex with PFKFB3 to induce glycolysis and cell cycle progression. This AGPG‐dependent oncogenic alteration contributes to the ESCC development and could be used as a therapeutic target for ESCC treatment
