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. 2020 Jul 17;11(7):544. doi: 10.1038/s41419-020-2731-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — In the initial experiments, immunofluorescence staining images demonstrated the localization of Vimentin (red) and E-cadherin (red) in the proximal tubular cells of kidneys of Control (a1-3), ZnT8-KO mice (b1-3), STZ-induced diabetic (c1-3) and ZnT8-KO-STZ mice (d1-3) and data are represented in a, b, respectively. AQP1 (green) is the marker for tubular cells. Original magnification, ×400. Data from Western blotting analyses were followed in C. Relative expression of Vimentin and E-cadherin was calculated and normalized to the loading controls. The experiments were repeated for three times (**P < 0.001 vs. Control mice and ^##P < 0.001 vs. STZ-induced diabetic mice).