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. 2020 Jul 21;40:18. doi: 10.1186/s41232-020-00118-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Role of TLR9 in the development of cardiometabolic disorders. DNA fragments and/or nuclear proteins released from damaged cells/tissues activate immune cells such as macrophages and DCs through TLR9, leading to the development of inflammation in these tissues, which is central in the pathogenesis of cardiometabolic disorders