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. 2020 Jul 22;11:313. doi: 10.1186/s13287-020-01834-0

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — iMSC-sEV treatment reduces tissue loss in the chronic stage of ischemic stroke and promotes long-term neurological recovery. a Representative images of T2-MRI scan (left two columns) performed on days 2 and 28, as well as MAP-2 immunostaining (Right column) on day 28 after MCAO. Dashed line (red and white): infarct border. CL, contralateral side. IL, ipsilateral side. Scale bar = 2 mm. b Quantification analysis of the percentage of infarct volume on day 2 and day 28 after MCAO. N = 8 rats per group. Behavior tests were performed by using mNSS score (c) and foot-fault test (d) before and up to 28 days after MCAO. N = 5 rats for sham group. N = 10 rats for vehicle and iMSC-sEV groups. Data are presented as mean ± SD. *P < 0.05. n.s indicates no significant difference