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. Author manuscript; available in PMC: 2020 Jul 22.
Published in final edited form as: Dev Cell. 2019 Feb 28;48(6):873–882.e4. doi: 10.1016/j.devcel.2019.02.003

Figure 2. Many kinetochore components are required for proper NMJ formation.

Figure 2.

(A-F) For the indicated genotypes, loss of a kinetochore protein causes overly long neurites (magenta arrows) and multiple small vesicle clusters stained with anti-CSP (blue arrows), and few or no large boutons (white arrows). (G) Schematic of the subcomplexes that make up the kinetochore. (H, I) Quantification of the phenotypes for kinetochore mutants and RNAi knockdown, colored to correspond to the subcomplexes in (G). Each genotype was compared with WT using one-way ANOVA with Dunnett’s multiple comparisons test. Scale bars, 10 μm. ***p<0.001, **p<0.01, *p<0.05. Error bars, mean ± s.e.m.