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. 2020 Jul 16;45(8):1839–1850. doi: 10.1007/s11064-020-03078-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Docosahexaenoic acid (DHA) played a protective role in TBI rats through Nrf2 signaling pathway. In this figure, a total of 100 rats were evenly divided into four groups: sham, TBI, TBI + DHA and TBI + vehicle (V). a Western blot (WB) and b quantitative real-time polymerase chain reaction (qRT-PCR) assays determined the expression levels of HO-1, NQO-1 and NADPH oxidase (NOX₂) in hippocampal tissues of rats in four groups. ***P < 0.001, *P < 0.05, vs. sham; ^#P < 0.05, ^##P < 0.01, ^###P < 0.001, vs. TBI + V