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. 2020 Jul 17;11:1065. doi: 10.3389/fphar.2020.01065

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Copyright © 2020 Xing, Fu, Cheng, Cai, Wang, Deng, Gong and Chen

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Hyperoside protection of CCl4-induced liver injury against oxidative damages in vivo. (A) Western blot. The expressions of total Fyn, HO-1, p-Nrf2 (S40), the cytoplasmic and nuclear p-Fyn and Nrf2 were analyzed respectively in liver tissues from animal experiments. (B) Representative quantification data of Fyn, HO-1, cytoplasmic Nrf2, cytoplasmic p-Fyn, nuclear Nrf2 and nuclear p-Fyn. These western blots were quantified by using Image J Software and the data are expressed as mean and standard deviation (SD) and statistically analyzed by using the ANOVA. ^aP < 0.05 vs. Control, ^bP < 0.05 vs. CCl4, ^cP < 0.05 vs. Hyp (60 mg/kg), ^dP < 0.05 vs. Hyp (15 mg/kg + CCl4), and ^eP < 0.05 vs. Hyp (30 mg/kg + CCl4).