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The oxysterol receptor GPR183 orchestrates ILC migration, lymphoid tissue formation and inflammatory responses in the colon. Oxysterols produced by fibroblastic stromal cells attract GPR183‐expressing ILCs to sites where lymphoid tissues (cryptopatches, isolated lymphoid follicles) are formed in the steady‐state colon. The oxysterol‐GPR183 pathway also controls the formation of inflammatory infiltrates during colitis.
