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. 2020 Jul 14;14(2):466. doi: 10.4081/oncol.2020.466

Table 2.

Summary of CD95/CD178 polymorphisms in hematological malignancies.

Type of malignancy Evaluated SNPs Number of Case/ Control Method Results Ref.
CD95 CD178
rs2234767 rs1800682 rs763110
(-1377G>A) (-670T>C) (-844T>C)
AML United Kingdom - 454/934 PCR-RFLP - CD95-1377AA has twice AML development risk. - 1377A/-670A haplotype of CD95 promoter SNP had highest risk of AML development. 18
Korea 592/858 PCR - Not associated with AML risk in the Korean population 19
China - - 98/2014 Meta-Analysis - Was not associated with AML developing. 20
Childhood de novo AML Multi-Ethnicity - - 440/ Not mentioned PCR - Reduction in CD95 enriches the expression of apoptosis resistant clones in AML blast cells. 25
Childhood ALL China - 361/519 PCR-RFLP - Combination of some protection variant forms is associated with decreased risk of childhood ALL 22
ALL Iran - 142/117 PCR-RFLP - –670 GG genotype is associated with liver involvement. 23
CML India - - 386/386 PCR-RFLP - CD95 −670 GG and CD178 −844 TC SNPs are associated with reduced event-free survival and development of CML
- CD95-670 AG associated with accelerated phase
- CD178-844TC SNP associated with development of blast phase
21
ATL Brazil - - 31/33 PCR-RFLP - Associated with susceptibility, clinical manifestation and survival. 24
FL Spain - - Cohort, 126 PCR - The C allele in this site is associated with a better response to rituximab therapy 26

AML, Acute myeloid leukemia; ALL, Acute lymphoblastic leukemia; CML, Chronic myeloid leukemia; ATL, adult T leukemia; FL, follicular lymphoma; SNP, single nucleotide polymorphism; PCR, polymerase chain reaction; PCR-RFLP, polymerase chain reaction-restriction fragment length polymorphism.