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. 2020 Jul 28;13:107. doi: 10.1186/s13041-020-00646-w

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Co-activation of IC and BLA neurons regulates taste memory allocation in the IC. a Experimental schema. b Data showing the mean quantity of saccharine solution consumed during the conditioning. c Data showing the aversion index in memory retrieval test (saline group, N = 16, CNO group, N = 17 animals). d Representative images showing expression of c-fos (green) after memory retrieval test and that of hM3Dq (magenta). Yellow arrows indicate double-labeled neurons (c-fos + and hM3Dq+). Scale bars = 100 um. e-f Probability of expression of c-fos in DAPI cells (e) and in the hM3Dq + neurons (f) in the IC and BLA (IC, saline group, N = 5, CNO group, N = 6 animals; BLA, saline group, N = 5, CNO group, N = 6 animals). Data are represented as mean ± SEM; *p < 0.05 and **p < 0.01 (saline-injected mice, black columns; CNO-injected mice, red columns)