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. Author manuscript; available in PMC: 2021 Jun 1.
Published in final edited form as: Atherosclerosis. 2020 May 22;303:36–42. doi: 10.1016/j.atherosclerosis.2020.04.020

Table 2.

The physiological role of major HDACs in the phenotype on vascular cells.

Subtypes Models Functions and phenotypes References

HDAC1 ECs Stimulates angiogenesis Kim M S (12)
HAECs Promote cell survival and prevent cell apoptosis Li Y (81)
HDAC2 SMC Inhibit cell proliferation Findeisen HM (49)
HAECs Inhibit vascular dysfunction Pandey D 82)
HDAC3 ECs Stimulate EC differentiation Zang L (17)
Macrophages Upregulate in atherosclerosis Hoeksema MA (85)
HDAC5 ECs Repress angiogenesis Urbich CL (22)
HUVECs Repress angiogenesis Ha CH (21)
HDAC6 Mice Increase blood pressure and vasoconstriction Chi Z (25)
SHR Increase vascular hyperplasia or vasoconstriction Choi SY (62)
ApoE-/- mice Develop atherosclerosis Leucker TM (90)
HDAC7 ECs Stimulate cell migration Mottet D(27)
Mouse Rupture of blood vessels Chang S (28)
HDAC8 SMCs The marker of smooth muscle differentiation Waltregny D (54)
HDAC9 Rats Increase EC permeability Shi W (38)
LDLr-/- mice Develop atherosclerosis Cao Q (94)
HDAC10 ECs Stimulate tube formation Duan B (39)
HDAC11 Rats Induce vessel injury Zhou B (40)
SIRT1 EC Modulate homeostasis Wan, Z (44)
Mice Inhibit vascular remodeling Gao P (70)
Mice Inhibit neointima formation Li L (71)
HUVECs Enhance EC survival Zhang QJ (87)
ECs Anti-inflammation Stein S (88)
SIRT3 SMCs Decrease proliferation Wu X (72)
SIRT6 ECs Prevent senescence Cardus A (48)

HAECs: human aortic endothelial cells; HUVECs: human umbilical vein endothelial cells. SHR: spontaneously hypertensive rat. ECs: endothelial cells: SMCs: smooth muscle cells.