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. 2020 Jul 21;11(7):e00201. doi: 10.14309/ctg.0000000000000201

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© 2020 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of The American College of Gastroenterology

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 2. — The forest plots of the comparison on virus prevalence between patients with gastric cancer and any controls after excluding subjects with known H. pylori and EBV positive: (a) the forest plot for HBV; (b) HCMV: Jin [1–5]: different sequences of HCMV genome were detected; Zhang [1–2]: IgG or IgM was detected; (c) the forest plot for HPV: Kamangar [1–3]:different subtypes of HPV were investigated; Ma [1–2]:different test methods were used; and (d) the forest plot for JCV: Jang [1–2]: different test methods were used; Murai [1–3]: different sequences of JCV genome were detected by Southern blot; Murai [4]: T-Ag were detected by immunohistochemistry; Shin [1–3]: different sequences of JCV genome were detected. CI, confidence interval; EBV, Epstein-Barr virus; HCMV, human cytomegalovirus; HPV, human papillomavirus; JCV, John Cunningham virus; OR, odds ratio; T-Ag, transforming antigen.