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. 2020 May 7;34(8):2074–2086. doi: 10.1038/s41375-020-0826-9

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — The left-side panel shows the impact of +8 (a), +Ph (b), complex ACA (c), and of chromosome 3, 7, 17, 19, and 21 aberrations combined (d) on survival in patients with primary imatinib treatment after the emergence of ACA. Suvival after emergence of low-risk ACA in imatinib-treated patients serves as control. The right-side panel shows the impact of rare high-risk ACA of chromosomes 3, 7, 17, 19, and 21 on survival (e–i).