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. Author manuscript; available in PMC: 2020 Jul 29.
Published in final edited form as: Horm Behav. 2020 Feb 29;121:104719. doi: 10.1016/j.yhbeh.2020.104719

Table 4.

Significantly enriched interactions of DEGs indicating the gene ontology and biological pathways (KEGG & Reactome) in WT males v. WT females.

Description FDR
GO: molecular function Histone demethylase activity 2.10845E–05
Steroid hormone receptor binding 0.003656028
Estrogen response element binding 0.006161726
Nuclear hormone receptor binding 0.006985698
Estrogen receptor activity 0.00720369
hormone receptor binding 0.00854285
GO: biological processes Histone demethylation 4.97867E–05
Regulation of macromolecule metabolic process 0.000254213
Regulation of phospholipase C activity 0.0008599
Regulation of lipase activity 0.003559273
Response to steroid hormone 0.004524849
Response to estrogen 0.004724675
Regulation of circadian rhythm 0.006080162
Intracellular steroid hormone receptor signaling pathway 0.007111892
Steroid hormone mediated signaling pathway 0.010956526
chromatin organization 0.012145584
Hormone-mediated signaling pathway 0.013819596
Negative regulation of triglyceride metabolic process 0.015261211
Cellular response to steroid hormone stimulus 0.017041363
Biological pathways (Reactome) HDMs demethylate histones 6.88327126245209E–07
Chromatin organization 1.42112E–06
RUNX1 regulates estrogen receptor mediated transcription 0.017513604
Biological pathways (KEGG) Transcriptional misregulation in cancer 0.002297158
Melanoma 0.004863766
Prostate cancer 0.006409001
Endocrine resistance 0.006409001
Thyroid hormone signaling pathway 0.008216614
Breast cancer 0.01121615
MicroRNAs in cancer 0.01121615