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. 2020 Jul 29;20:350. doi: 10.1186/s12935-020-01402-9

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — FOXD3-AS1 promoted chemo-resistance in NSCLC cells. a qRT-PCR determination of FOXD3-AS1 in cell lines including NHBE, A549, H1299, A549/DDP and H1299/DDP. b qRT-PCR determination of FOXD3-AS1 in A549 and H1299 cells with pcDNA3.1 or pcDNA3.1-FOXD3-AS1 transfection. c–e CCK-8 assay determination of IC50 values (cisplatin) in A549 and H1299 cells with pcDNA3.1 or pcDNA3.1-FOXD3-AS1 transfection. f qRT-PCR determination of FOXD3-AS1 in A549 and H1299 cells with si-NC or si-FOXD3-AS1 transfection. g–i CCK-8 assay determination of IC50 values (cisplatin) in A549 and H1299 cells with si-NC or si-FOXD3-AS1 transfection. N = 3 biological samples, and each sample was assayed in triplicates. Significant different between different treatment groups were shown as *P < 0.05 and **P < 0.01