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Scheme showing potential relationship among AA, PGE2, LXA4 and viral load in a COVID-19 patient who succumbs to the disease. Note the absence of biphasic nature of AA release and failure of PGE2 to reach its peak and as a result deficiency in LXA4 synthesis occurs that results in failure of resolution of inflammation (compare with Fig 1). DGLA, EPA and DHA (not shown in the figure) may have actions/functions like AA.
