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. 2020 Jul 29;11(7):594. doi: 10.1038/s41419-020-02784-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a FISH assay was used to determine the location of circATRNL1, miR-141-3p, and miR-200a-3p in Ishikawa cells (blue, DAPI nuclear staining; red, circATRNL1; green, miR-141-3p and miR-200a-3p). Scale bars represent 20 μm. b The binding sites between wild-type circATRNL1 or mutated circATRNL1 and miR-141-3p/miR-200a-3p were predicted by using bioinformatics analysis. c Dual-luciferase reporter assays were carried out in HEK-293T cell to confirm the combination between circATRNL1 and miR-141-3p mimics or miR-200a-3p mimics. d qRT-PCR analysis was carried to determine the expression pattern of miR-141-3p and miR-200a-3p in ovary endometriosis tissues. ****P < 0.0001 represents statistical difference.