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. 2020 Jul 30;11(7):605. doi: 10.1038/s41419-020-02807-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Cells simultaneously underwent transfection with si-circ9119 and an miR-26a inhibitor (or NC inhibitor) for 36 h. a, b qRT-PCR analysis showing miR-26a expression in each group. c, d MTT assay showing HepG2 and Huh-7 proliferation rates, under different transfection conditions. e, f Colony formation assay indicating HepG2 and Huh-7 cell growth rates, under various transfection conditions. g, h FCM analysis showing the number of apoptotic cells following different treatments of HepG2 and Huh-7 cell lines. *p < 0.05 vs. indicated groups.