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. 2020 Jul 30;11(7):605. doi: 10.1038/s41419-020-02807-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — TNM3 silencing reduces proliferation of SW579 cells. Cells were incubated with 2 μM baricitinib or 1% DMSO for 1 h. a, b Western blot analyses showing JAK1 and STAT3 expression, and STAT3 phosphorylation in HepG2 and Huh-7 cells, following incubation with baricitinib or DMSO. c, d Immunofluorescence assay showing the cellular localization of STAT3 in cells after incubation with baricitinib or DMSO. e, f Colony formation assay indicating HepG2 and Huh-7 growth rates, with baricitinib or DMSO treatment. g, h FCM evaluation of HepG2 and Huh-7 cell apoptosis with baricitinib or DMSO treatment. *p < 0.05 vs. indicated groups.